Blood Types of Abductees, Experiencers and Contactees: What Could They Indicate?
By Richard Bonenfant, Ph.D.
“An ongoing attempt is being made by abduction researchers to determine the possible significance of the Rh O- blood type among experiencers. This effort also attempts to determine possible attributes associated with this blood type.”
If O− blood is nothing more than a mutation, then why?... why do people think a mutation made it that way? What are the odds that DNA not only mutated to that extent but also mutated to such a useful degree?
The essence of this compound question is based upon two, as yet, unproven assumptions: that the Rh O- blood type is an exceptional blood line and that its expression confers exceptional abilities.
From a scientific perspective, ABO blood types have been present since before humans diverged from our simian ancestors millions of years ago in Africa. It’s true that geneticists have yet to determine the precise origin of Rh O-. In consequence, numerous hypotheses have been advanced to account for its presence in the Human genome. These hypotheses range from a random genetic mutation in the O blood type to its origin from ancient Jewish or Basque populations to its deliberate insertion by the Anunnaki or other alien agencies. . Because of its high frequency in northern Europeans, one of the most current hypotheses is the Rh O blood types conferred to its possessors breathing benefits in colder latitudes.
While scientists have no idea how the Rh O- blood line arose, they support the view that it arose from errors in protein replication. The protein chains that make up our genes can be likened to a single card within a deck of cards (the gene). Depending on which card is affected, it could negatively affect the outcome of the deck’s purpose.
Nearly all major mutations or replication errors are dysfunctional and consequently abort the fetus during gestation. A small number of mutations are either neutral or potentially positive in effect. Those positive mutations which confer a survival or reproductive advantage to a species tend to proliferate within the species over time. Given the pace of recent advances in genetic sequencing, it’s likely that the mystery of Rh O- blood type origin and benefits will be uncovered in the very near future.
 Origin Theories of the Rare Rh-Negative Blood Factor, The Rh-Negative Registry.
O Negative Blood is Not a Mutation
An argument has been advanced that Rh O- blood type is not a mutation and is unrelated to the other Rh antigen groups. This hypothesis suggests that the Rh negative variant of the Rh O+ blood line was not the result of natural mutation. I understand this view to be; the rarity of the Rh O- blood type coupled with its reputed status as a universal donor for transfusion implies that it was deliberately introduced into the human genome by some unknown agency.
Since the origin of this bloodline has not as yet been defined by geneticists and because of its extreme rarity in the population, the view that Rh O- is a non-mutational artifact cannot be dismissed. However, despite its importance as being the universal donor, other benefits attributed by this bloodline remain in question. The genetic origin of human blood types is presently in a state of flux as new research uncovers rare exceptions to the traditional ABO and Rh classification. [2,3] Whether the Rh O- blood type is derived from a random mutation or not remains to be determined. The argument that it is a deliberate modification of our genetic structure cannot be ruled out because our society is presently on the cusp of genetic engineering. However, the consensus of most geneticists is that the Rh O- blood type will be found to be a natural mutation.
 “A total of 33 human blood group systems are now recognized by the International Society of Blood Transfusion (ISBT). The two most important ones are ABO and the RhD antigen”
 Blood mystery solved: Two new blood types identified, Science Daily, February 23, 2012.
Summary: “You probably know your blood type: A, B, AB or O. You may even know if you’re Rhesus positive or negative. But how about the Langereis blood type? Or the Junior blood type? Positive or negative? Most people have never even heard of these. Yet this knowledge could be “a matter of life and death.” While blood transfusion problems due to Langereis and Junior blood types are rare worldwide, several ethnic populations are at risk.”
An ongoing attempt is being made by abduction researchers to determine the possible significance of the Rh O- blood type among experiencers. This effort also attempts to determine possible attributes associated with this blood type. To date, the results of these efforts have been tantalizing but not definitive. A larger collection of experiencers would greatly help define the study’s outcome. However, regardless of the presence of a correlation, it is vital to continue efforts of this type. The abduction community must unite in their efforts to understand the nature of this phenomenon by advancing and supporting research similar to that presented above. It’s presently doubtful that the scientific community will respect this effort until we are able to speak their language. Regardless of the outcome of these efforts, it is fully evident to abductees that their experiences are true and valid. Therefore, in spite of scientific neglect, abductees and those who find merit and trust in their testimonies must continue efforts to investigate this disturbing phenomenon.
Further Reading Regarding Rh Factor
“Antigens of the Rh blood group system are products of RHD and RHCE (collectively referred to as RH30 or RHCED), two tightly linked and highly homologous genes residing on chromosome 1p36.1. RhD carries the D antigen, the most potent blood group immunogen. The D epitope is not expressed in a relatively large segment of the population (i.e., Rh-negative phenotype), as a result of RHD gene deletion or other gene alterations.” Source
The Rh blood group antigens derive from 2 genes, RHD and RHCE, that are located at chromosomal position 1p34.1-1p36 (chromosome 1, short arm, region 3, band 4, subband 1, through band 6). In whites, a cde haplotype with a deletion of the whole RHD gene occurs with a frequency of approximately 40%. The relative position of the 2 RH genes and the location of the RHD deletion was previously unknown. A model has been developed for the RH locus using RHD- and RHCE-related nucleotide sequences deposited in nucleotide sequence databases along with polymerase chain reaction (PCR) and nucleotide sequencing. The open reading frames of both RH genes had opposite orientations. The 3’ ends of the genes faced each other and were separated by about 30 000 base pair (bp) that contained the SMP1 gene. The RHD gene was flanked by 2 DNA segments, dubbed Rhesus boxes, with a length of approximately 9000 bp, 98.6% homology, and identical orientation. The Rhesus box contained the RHD deletion occurring within a stretch of 1463 bp of identity. PCR with sequence-specific priming (PCR-SSP) and PCR with restriction fragment length polymorphism (PCR-RFLP) were used for specific detection of the RHD deletion. The molecular structure of the RH gene locus explains the mechanisms for generating RHD/RHCE hybrid alleles and the RHD deletion. Specific detection of the RHD(-) genotype is now possible. (Blood. 2000;95:3662-3668) Source: Wagner FF, Flegel WA (Aug 2000). “RHD gene deletion occurred in the Rhesus box”. Blood 95 (12): 3662–8. PMID 10845894.
“Another definition of null allele concerning molecular markers, refers to such a marker in the case it can no longer be detected because of a mutation. For example, micro satellites (i.e. a repetitive sequence of DNA, in which the repeat is rather short) are used as molecular markers amplifying them through PCR. To do so, a primer or oligonucleotide aligns with either of ends of the locus. If a mutation occurs in the annealing site, then the marker can no longer be used and the allele is turned into a null allele.”
“One example of a null allele is the ‘O’ blood type allele in the human A, B and O blood type system. The alleles for the A-antigen and B-antigen are co-dominant, thus they are both phenotypically expressed if both are present. The allele for O blood type, however, is a mutated version of the allele for the A-antigen, with a single base pair change due to genetic mutation. The protein coded for by the O allele is enzymatically inactive and therefore the O allele is expressed phenotypically in homozygous OO individuals as the lack of any blood antigen. Thus we may consider the allele for the O blood type as a null allele.” Source © GETR/AJ